Explanation Process and Knowledge Revision in the Light of Discovery of Cardioprotection Phenomenon
Abstract
The purpose of the present paper is to analyze the knowledge revision that occurred in the process of explanation an abnormal phenomenon in cardiology. S. Bromberg’s description of abnormic laws will provide the point of departure for the present analysis. It will be shown here that in the example the revision of knowledge resulted in formulating an abnormic law that deviated from the empirical law in cardiology. T. Kuipers’ and A. Wisniewski’s ideas of explanation by specyfication will be used for reconstructing the example in question
References
Bromberger S. [1966], Why Questions, [w:] R.G. Colodny (ed.), Mind and Cosmos, Pittsburg: University of Pittsburgh Press, s. 86-111.
Geft I., Fishbein M., Ninomiya K., Hashida J. (et al.) [1982], Intermittent brief periods of ischemia have a cumulative effect and may cause myocardial necrosis, „Circulation” 66, s. 1150-1153.
Grobler A. [2006], Metodologia Nauk, Kraków: Wydawnictwo Aureus – Wydawnictwo Znak.
Grobler A., Wiśniewski A. [2005], Explanation and Theory Evaluation, „Poznań Studies in the Philosophy of the Sciences and the Humanities”, vol. 84, s. 299-310.
Kuipers T., Wiśniewski A. [1994], An Erotetic Approach to Explanation by Specification, „Erkenntnis” 40, s. 377-402.
Lange R., Ware J., Kloner R. [1984a], Absence of a cumulative deterioration of region al function during three repeated 5 or 15 minute coronary occlusions, „Circulation” 69, s. 400-408.
Lange R., Ingwall S., Hale S., Alker K., Kloner R. [1984b]: Effects of reccurent ischemia on myocardial high energy phophate content in canine hearts, „Basic Research in Cardiology” 79, s. 469-478.
Lipton P. [2004]: Inference to the Best Explanation, London–New York: Routledge.
Murry C., Jennings R., Reimer K. [1986], Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium, „Circulation” 74, s. 1124-1136.
Reimer K., Jennings R. [1979], The wavefront phenomenon of myocardial ischemic cell death. II. Transmural progression of necrosis within the framework of ischemic bed size (myocardial at risk) and collateral flow, „Laboratory Investigation” 40, s. 633-644.
Reimer K., Murry C., Yamasawa I. (et al.) [1986], Four brief periods of myocardial ischemia cause no cumulative ATP loss or necrosis, „American Journal of Physiology”, 251, 1306-1315.
Rzepiński T. [2007], The Structure of Diagnosis In Medicine: Introduction to Interrogative Characteristics, „Theoretical Medicine and Bioethics” 28, s. 63-81.
Rzepiński T. [2009], Metodologiczne aspekty badań eksperymentalnych w rozwoju strategii kardioprotekcyjnej. Część II: Od hartowania niedokrwiennego do postconditioningu”, „Nowiny Lekarskie” 2(78), s. 135-147.
Swain J., Sabina R., Hines J. (et al.) [1984], Repetitive episodes of brief ischaemia (12 min) do not produce a cumulative depletion of high energy phosphate compounds, „Cardiovascular Research” 18, 264-269.
Wiśniewski A. [1999], Erotetic Logic and Explanation by Abnormic Hypotheses, „Synthese” 120, s. 295-309.
Wiśniewski A. [2001], Questions and Inferences, „Logique & Analyse” 173-174-175, s. 5-43.
Copyright (c) 2011 Roczniki Filozoficzne
This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.
